Ovar cancer and their consequences
Introduction
Various investigations report that the pathophysiology of this disorder is not entirely clear, it is given that in large part hyperandrogenism is a consequence that there is no stability between the ability to make a reserve of lipids that the woman possesses. The determination is announced thanks to abiogenesis in early periods that happen in life and also thanks to the amount or number of lipids, that is, the caloric contribution which should have a storage during times that go later in life.
When there is a situation in which an exaggerated or excessive caloric contribution is presented, lipids are normally located in different and liver in the liver. This is defined whether or not there is a presence of insulin resistance, increased synthesis that may occur of androgens and gonadotropins, in addition to a moment of inflammation of decreased level.
Developing
The prevalence of obesity in young children and the small weight at the time of birth going to hand with a speed and aggressive postnatal growth is required in predisposing components in the progress of this chain in patients with genetic delicacy. Today there is no count about the diagnostic criteria of this disorder during adolescence.
Being able to use the criteria allowed in women, a diagnosis that is bad can be established, usually happens mostly in ladies who occur to them irregularity in their menstrual cycles in the initial years of post-metharquia results with physiological aspects. As there is no presence of a count, in this study the suggestion of this triad was made for the clinical part:
That there is no absence of hirsutism or inflammatory acne, also the increase in total testosterone or in the index of free androgens in its concentrations, during the follicular phase of the menstrual cycle that happens between the third to the eighth day, it can also be after 2 months In the presence of amenorrhea, there is also the oligomenorrhea that is defined as the presence of cycles greater than forty five days or also the secondary amenorrhea that is known as the menstrual fault that happens greater than three months, 2 years or more posterior of the menarche.
It can be said that 2/3 of women with this disorder of polystic ovaries have an obvious insulin resistance, and is announced through glucosuric basal concentrations and insulin thanks to the “homeostasis evaluation model” (Homa ). Insulin resistance is announced due to the lack of obesity, something that helps in the centripetal distribution of fat always focusing on the visceral fat that is made known in these young women, is related to a lipid profile that does not provide help to help favor it and also brings various changes and alters inflammation indicators (extra sensitive C -reactive protein), and possible cardiovascular problem.
conclusion
During the youth that suffers from obesity, also in adolescents who had a low weight at the time of birth, or puberty occurred from before and menarche equal since before, it would be appropriate for a realization to add an oral glucose overload, everything for a large risk factor to suffer from glucose intolerance. There is an establishment with the differential diagnosis in relation to other diagnoses, mainly with congenital adrenal hyperplasia due to lack of hydroxilli. This is why there is an establishment that is systematically carried out in serum hydroxiprogesterone concentrations.
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